西安鲲创科技发展有限公司长期致力于高脂细胞添加剂的研发工作，产品涉及软脂酸/硬脂酸/棕榈酸钠/棕榈酸/油酸钠/亚油酸/月桂酸/豆蔻酸/亚麻酸/花生四烯酸等高脂细胞添加剂，具有溶剂无毒 、无固体析出、浓度准确、 液态即用、可长期保存等核心优势。产品可用于诱发细胞脂毒性、内质网应激、氧化应激、线粒体损伤、细胞凋亡等，模拟高脂环境对人体细胞的损害。目前，已经积累了上千用户，用户群涉及清华大学、北京大学、浙江大学、武汉大学基础医学院、同济大学、第二军医大学、第四军医大学、新疆医科大学、昆明理工大学、武汉市协和医院、中科院亚热带所等全国范围的高校、研究所和医院以及企业，仅在丁香园发出的交流贴也受到了100000+的关注量。在我司产品和技术支持下，用户已经完成了多篇SCI论文，其中包括Science Advances、Diabetes等糖尿病领域顶级期刊，已发表的SCI、Nature很多，您谷歌学术搜索“Kunchuang Biotechnology”（鲲创生物）即可如下：     

  [1] Gao W ,  Guo X ,  Wang Y , et al. Monocyte-derived extracellular vesicles upon treated by palmitate promote endothelial migration and monocytes attachment to endothelial cells[J]. Biochemical and Biophysical Research Communications, 2020, 523( 3):685-691.
   [2]Liu, Fengzhou, Li, et al. MICU1 Alleviates Diabetic Cardiomyopathy Through Mitochondrial Ca2+-Dependent Antioxidant Response[J]. Diabetes A Journal of the American Diabetes Association, 2017.
   [3] Yan W ,  Zhang H ,  Liu P , et al. Impaired mitochondrial biogenesis due to dysfunctional adiponectin-AMPK-PGC-1α signaling contributing to increased vulnerability in diabetic heart[J]. Basic Research in Cardiology, 2013, 108(3):329.
   [4]Targeting ferroptosis suppresses osteocyte glucolipotoxicity and alleviates diabetic osteoporosis[J]. Bone Research.
   [5] Cardiac-specific CGI-58 deficiency activates the ER stress pathway to promote heart failure in mice[J]. Cell Death & Disease.
   [6] Chen S ,  Che S ,  Li S , et al. The combined impact of decabromodiphenyl ether and high fat exposure on non-alcoholic fatty liver disease in vivo and in vitro[J]. Toxicology, 2021, 464:153015.